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Murray Edwards College
University of Cambridge
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    Dr Julia Turner

    Murray Edwards College
    New Hall
    University of Cambridge
    Cambridge
    Cambridgeshire
    CB3 0DF

    Bye Fellow in Pathology

    Murray Edwards has been a very significant part of my life for over 30 years, as an alumna and academic member. The College community has continued to evolve in response to the enormous changes in women's lives and to strengthen its unique contribution to women's education. Women are so happy truly realising their potential here.

    Degrees and honours

    • BA
    • MA
    • PhD.

    Research interests

    Dr Turner's laboratory in the Pathology Department, and subsequently at Glaxo Wellcome, focused on the molecular control of the division of white blood cells - understanding this is key both to our ability to increase a desirable immune response (as in immunisation) or reduce a pathological one (as in autoimmune disease and allergy).

    Biography

    After reading Natural Sciences (Part II Pathology) here at New Hall, Dr Julia Turner completed her PhD in Molecular Oncology at The Ludwig Institute for Cancer Research in Cambridge before moving to the University of California, San Francisco to study signal transduction pathways involved in T cell activation. After working with Dr Tim Hunt (Imperial Cancer Research Fund) to learn about the control of the cell cycle, she became a Cambridge University Lecturer in Immunology in the Department of Pathology, her lab focusing on the molecular biology of T cell division. She became a Fellow of New Hall in 1991. She experienced research in the private sector and then returned to San Francisco for several years. She returned to Cambridge as a wife and mother and continue to love her role as a Bye Fellow and supervisor in Pathology to all the Medical, Veterinary and Natural Sciences students.

    Publications

    • Fehérvári, Z., Cooke, A., Brett, S. and Turner, J. (2002). Perturbation of naive TCR transgenic T cell functional responses and upstream activation events by anti-CD4 monoclonal antibodies. Eur. J. Immunol., 32: 333–340
    • Turner, JM (1993). IL-2-dependent induction of G1 cyclins in primary T cells is not blocked by rapamycin or cyclosporin A. International immunology, 5(10), 1199-1209.
    • Glaichenhaus, N., Shastri, N., Littman, D.R. & Turner, JM (1991) Requirement for association of p56lck with CD4 in antigen-specific signal transduction in T cells. Cell 64, 511-520. 
    • Turner, JM Brodsky, M.H., Irving, B.A., Levin, S.D., Perlmutter, R.M. & Littman, D.R. (1990) Interaction of the unique amino-terminal region of the tyrosine kinase p56lck with the cytoplasmic domains of CD4 and CD8 is mediated by cysteine motifs. Cell 60, 755-765
    • Ibson JM,  Rabbitts PH (1988) . Sequence of a germ-line N-myc gene and amplification as a mechanism of activation. Oncogene 2(4):399-402.